To Becky and all above,Becky I felt the same way when my normal hehtlay 7yr old was diagnosed with type 1D. It was really hard for me to except. It has been 4yrs and I still read and research everything I can. I do have many concerns. 1st is genetics, we have autoimmune diseases in our family such as Hypothyroid disease and Rheumatoid Arthritis. 2nd is, there is a test that can be given at the age of 5yrs that will alert Drs. if a child is high risk. Why isn’t it a required test? 3rd is environmental factors. Why haven’t they been identified and made public? 4th is, the way Drs. train us to care for our children after diagnoses. During onset we are told to administer insulin based on carbs. and to push carbs. because the body needs it to develop. I’m ok with that, but more carbs. means more insulin which means the Drs. and Pharmaceutical Com. make money. 5th if we push carbs. like this we are creating fat cells in our children that will lead to obesity and more health problems.The food that our children consume and the medical practices of today make it hard to believe that anyone really cares about anything except the almighty $$$$. 6th is vaccines, My sons onset took place after a really bad reaction to the DTAP and several bouts of viral throat infections.My son is now going through puberty,and let me tell you that it is very demanding on the childs body. To the tune of 1u. of insulin per 4g. of carbs. That is alot of insulin.So we are now looking at other things like low carb. hi protein diets. And trying to cut carbs. to reduce the intake of insulin. If we cont. to do the work for the body it will never work for itself. Here is some info I found on the internet and I will include the link. Best of luck. And remember. You know more about your child than anyone else! You can choose to try other things. But never,never stop the shots until the blood sugars show that a reduction of insulin is needed. And pray hard for a cure.God Bless you and take care. Most cases (95%) of type 1 diabetes mellitus are the result of environmental factors interacting with a genetically susceptible person. This interaction leads to the development of autoimmune disease directed at the insulin-producing cells of the pancreatic islets of Langerhans. These cells are progressively destroyed, with insulin deficiency usually developing after the destruction of 90% of islet cells.
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